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The full picture is more nuanced than a simple angiogenesis equals cancer risk framing
human relevance uncertain)
DOI: 10.1038/s43587-023-00441-x
Hyperglycemia-driven AGE-RAGE signaling, PKC activation, and RAAS dysregulation converge on oxidative stress, endothelial dysfunction, and profibrotic transcription (e.g., TGF-beta/Smad), while mitochondrial and endoplasmic-reticulum stress amplify lipotoxicity and cell death
Moreover, upon SEMA treatment D-hpCAFs displayed decreased Col1A1 and P4HA1 expression levels (Suppl
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